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Disclosures: Wajiha Khan: Nothing to Disclose, David Kim: Nothing to Disclose, Ariana Shari: Nothing to Disclose, Hamza Khan: Nothing to Disclose, Alexander Le: Nothing to Disclose, Christopher Hawryluk: Nothing to Disclose, Ivan Bobkov: Nothing to Disclose, Ahmed Al-Khazraji: Nothing to Disclose, Kaveh Hajifathalian: Nothing to Disclose 1083 IMPROVED LIVER FIBROSIS REGRESSION AFTER DIRECT-ACTING ANTIVIRAL THERAPY IN HEPATITIS C PATIENTS WITH METABOLIC DYSFUNCTION-ASSOCIATED STEATOTIC LIVER DISEASE Alexis McCary 1 Yi-shin Sheu 1 Karen Chesbrough 1 Cabell Jonas 1 , 1 Mid-Atlantic Permanente Medical Group Background: One third of the US population has some degree of liver steatosis, and many patients with Chronic Hepatitis C (CHC) meet criteria for metabolic dysfunction-associated steatotic liver disease (MASLD)

Biochemistry 37:1792317930 van Dalen CJ, Whitehouse MW, Winterbourn CC, Kettle AJ (1997) Thiocyanate and chloride as competing substrates for myeloperoxidase

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As shown in Figure 9, most of the SlUGT genes, except for SlUGT23 , SlUGT48 , SlUGT72 , and SlUGT113 , could be expressed in tissue sites